In today’s ID the Future, intelligent design pioneer Michael Behe continues his conversation with philosophers Pat Flynn and Jim Madden. Here in Part 2 of a three-part series, Behe offers an illustration from language and Madden presses him, noting that meaning detection in language is not parts to whole. A lively exchange ensues and then Behe turns the discussion back to his primary focus, detecting design in molecular biological machines by recognizing the purposeful arrangement of parts. From there the conversation turns to everything from epigenetics, systems biology, and autopoiesis to co-option, mousetraps, tie clips, biologist Kenneth Miller, and the philosophers Aristotle and Thomas Aquinas. For Behe’s newest book, A Mousetrap for Darwin, go here. This discussion is presented here with permission of philosopher and podcaster Pat Flynn.
Today’s ID the Future concludes our series on A Mousetrap for Darwin, Lehigh University biochemist Michael Behe’s new book on evolution and intelligent design. Here Behe and host Eric Anderson tackle an objection to Behe’s work from evolutionary biologist Larry Moran. Moran says that while the Darwinian process may find it difficult to find any particular solution requiring evolutionary innovation, there are countless possible solutions to a given problem, not just the one solution that evolution did hit upon and that is under investigation. According to Moran, Behe failed to take this into account, a factor that greatly enhances the chances of blind evolution to engineer novel solutions to ecological challenges. Behe counters that Moran’s objection misses the force of the evidence gained from the study of evolution in the malaria parasite and in other microbes. That evidence shows that evolution is extremely limited in what it can achieve, no holds barred, no possible solutions disallowed. Behe also discusses recent research confirming Dollo’s Law, why that’s bad news for Darwinism, and why Behe’s time-symmetric Dollo’s Law spells even bigger trouble for Darwinism.
On this episode of ID the Future, biochemist Michael Behe and host Andrew McDiarmid discuss the anti-malarial drug chloroquine, now being investigated as a treatment for COVID-19, and how it may work on the cellular level against the coronavirus. The same drug was featured in Behe’s 2007 book The Edge of Evolution, as part of his demonstration that evolution has strict limits: It can do adaptive work for organisms with single mutations, but if just two coordinated mutations are required at once, evolution’s random processes have great difficulty even with natural selection helping them along. In cases where population sizes are enormous, as with malaria, it can eventually overcome the need for two simultaneous and coordinated mutations, but only just barely. Because the odds go up exponentially, three simultaneous coordinated mutations may be beyond the edge of evolution. What does all this bode for chloroquine and the coronavirus? Listen in as McDiarmid and Behe discuss.
On this episode of ID the Future, Dr. Michael Behe talks with Casey Luskin about recent findings that support his argument in The Edge of Evolution. Dr. Behe explains why Chloroquine, a drug that treats malaria, presents a good opportunity to study the limits of random mutation and natural selection, and how his conclusions inspired so much backlash — including misrepresentation of his argument — from his critics.